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NAFIS CERCULA HERB 17

NAFIS CERCULA HERB 17

herbs for arteriosclerosis


ID: 17


Pack Weight: 50 g.


Price: 42 $        




ingredients:

- Rosmarinus officinalis.
- Hyssopus officinalis and buds.
- Ginkgo Biloba.
- Melilotus officinalis.


Indications:

Indications:
This product control inflammatory mechanisms couple dyslipidaemia to atheroma formation. And leukocyte recruitment and expression of pro-inflammatory cytokines characterize early atherogenesis.


Atherosclerosis: over view
Atherosclerosis, the principal cause of heart attack, stroke and gangrene of the extremities, is responsible for 50% of all mortality in the USA, Europe and Japan. The lesions result from an excessive, inflammatory-fibroproliferative response to various forms of insult to the endothelium and smooth muscle of the artery wall. A large number of growth factors, cytokines and vasoregulatory molecules participate in this process. Our ability to control the expression of genes encoding these molecules and to target specific cell types provides opportunities to develop new diagnostic and therapeutic agents to induce the regression of the lesions and, possibly, to prevent their formation.

The pathogenesis of atherosclerosis is hypothesized to occur as a response to various forms of injury to the lining arterial endothelial cells. The resulting endothelial alterations could potentially lead to interactions between platelets in the circulation and the underlying subendothelial connective tissue or with the altered endothelial cells themselves. Such interactions provide an opportunity for platelet degranulation and release of a platelet-derived growth factor. This factor has been shown in cell culture to be an extremely potent mitogen and will induce DNA synthesis and cell multiplication of a number of cells including smooth muscle cells, fibroblasts, and other mesenchymally derived cells. Chronic endothelial injury and repeated interactions between platelet-derived mitogens, plasma components, and the underlying arterial smooth muslce cells would promote the progression of the intimal proliferative lesions of atherosclerosis that lead to the clinical sequelae associated with this disease process.

Inflammation in atherosclerosis:
Abundant data link hypercholesterolaemia to atherogenesis. However, only recently have we appreciated that inflammatory mechanisms couple dyslipidaemia to atheroma formation. Leukocyte recruitment and expression of pro-inflammatory cytokines characterize early atherogenesis, and malfunction of inflammatory mediators mutes atheroma formation in mice. Moreover, inflammatory pathways promote thrombosis, a late and dreaded complication of atherosclerosis responsible for myocardial infarctions and most strokes. The new appreciation of the role of inflammation in atherosclerosis provides a mechanistic framework for understanding the clinical benefits of lipid-lowering therapies. Identifying the triggers for inflammation and unravelling the details of inflammatory pathways may eventually furnish new therapeutic targets.

Sometimes lifestyle changes, such as quitting smoking, losing weight, eating a healthy diet and exercising, can help to control arteriosclerosis.


administration:

middle spoon , or tea bag from the mixture to a glass of boiled water /capacity of 200 ml. ( boiled previously) then cover it for ten minutes, filter it and have it warm half an hour before the meal.
In therapy cases: 2-3 times daily.
Prophylactically: once daily.


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